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MYCing and YAPing the escape of tumor cell growth arrest after chronic PI3K/mTOR inhibition

   
@article{JTD13404,
	author = {Georgios D. Vavougios and Sotirios G. Zarogiannis},
	title = {MYCing and YAPing the escape of tumor cell growth arrest after chronic PI3K/mTOR inhibition},
	journal = {Journal of Thoracic Disease},
	volume = {9},
	number = {5},
	year = {2017},
	keywords = {},
	abstract = {The phosphatidylinositol-3-kinase (PI3K)/Akt and mammalian target of rapamycin (mTOR) signaling cascades represent two critical and highly interconnected pathways regulating cellular proliferation, metabolism, differentiation and survival—particularly in response to cytotoxic stress (1). As a single pathway exploited by multiple neoplasms, mutations of PI3K/mTOR’s components represent attractive pharmacological targets, leading to the development of a wide array of targeted therapies (2).},
	issn = {2077-6624},	url = {https://jtd.amegroups.org/article/view/13404}
}